Core temperature and melatonin circadian rhythm period lengths are longer in delayed sleep phase disorder patients than controls

Gorica Micic; Nicole Lovato; Michael Gradisar; Sally Ferguson; Helen Burgess; Leon Lack

doi:10.1111/jsr.12213

Core temperature and melatonin circadian rhythm period lengths are longer in delayed sleep phase disorder patients than controls

Gorica Micic, Nicole Lovato, Michael Gradisar, Sally Ferguson, Helen Burgess, Leon Lack

Research output: Contribution to journal › Meeting Abstract › peer-review

Abstract

Delayed Sleep Phase Disorder (DSPD) is assumed to be caused by a persistent 2–6 h delay in the timing of patients’ biological clocks. Another mechanism, the endogenous circadian rhythm period lengths (i.e., taus) of core temperature and melatonin may contribute to the maintenance of the phase delay and resistance
to effective treatment. It was hypothesized that DSPD patients would display significantly longer taus compared to controls, thus contributing to its aetiology.

Original language	English
Article number	150
Pages (from-to)	34
Number of pages	1
Journal	Journal of Sleep Research
Volume	23
Issue number	s1
DOIs	https://doi.org/10.1111/jsr.12213
Publication status	Published - 8 Sept 2014
Event	22nd Congress of the European Sleep Research Society - Tallin, Estonia Duration: 16 Sept 2014 → 20 Sept 2014

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title = "Core temperature and melatonin circadian rhythm period lengths are longer in delayed sleep phase disorder patients than controls",

abstract = "Delayed Sleep Phase Disorder (DSPD) is assumed to be caused by a persistent 2–6 h delay in the timing of patients{\textquoteright} biological clocks. Another mechanism, the endogenous circadian rhythm period lengths (i.e., taus) of core temperature and melatonin may contribute to the maintenance of the phase delay and resistanceto effective treatment. It was hypothesized that DSPD patients would display significantly longer taus compared to controls, thus contributing to its aetiology.",

author = "Gorica Micic and Nicole Lovato and Michael Gradisar and Sally Ferguson and Helen Burgess and Leon Lack",

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AU - Micic, Gorica

AU - Lovato, Nicole

AU - Gradisar, Michael

AU - Ferguson, Sally

AU - Burgess, Helen

AU - Lack, Leon

PY - 2014/9/8

Y1 - 2014/9/8

N2 - Delayed Sleep Phase Disorder (DSPD) is assumed to be caused by a persistent 2–6 h delay in the timing of patients’ biological clocks. Another mechanism, the endogenous circadian rhythm period lengths (i.e., taus) of core temperature and melatonin may contribute to the maintenance of the phase delay and resistanceto effective treatment. It was hypothesized that DSPD patients would display significantly longer taus compared to controls, thus contributing to its aetiology.

AB - Delayed Sleep Phase Disorder (DSPD) is assumed to be caused by a persistent 2–6 h delay in the timing of patients’ biological clocks. Another mechanism, the endogenous circadian rhythm period lengths (i.e., taus) of core temperature and melatonin may contribute to the maintenance of the phase delay and resistanceto effective treatment. It was hypothesized that DSPD patients would display significantly longer taus compared to controls, thus contributing to its aetiology.

U2 - 10.1111/jsr.12213

DO - 10.1111/jsr.12213

M3 - Meeting Abstract

SN - 0962-1105

VL - 23

SP - 34

JO - Journal of Sleep Research

JF - Journal of Sleep Research

IS - s1

M1 - 150

T2 - 22nd Congress of the European Sleep Research Society

Y2 - 16 September 2014 through 20 September 2014

ER -

Core temperature and melatonin circadian rhythm period lengths are longer in delayed sleep phase disorder patients than controls

Abstract

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