Hyponatraemia in heart failure

Su Ling Tee; Andrew Sindone; Simon Roger; John Atherton; John Amerena; Michael D'Emden; Jonathan Erlich; Carmine De Pasquale

doi:10.1111/imj.14624

Hyponatraemia in heart failure

Su Ling Tee, Andrew Sindone, Simon Roger, John Atherton, John Amerena, Michael D'Emden, Jonathan Erlich, Carmine De Pasquale

Research output: Contribution to journal › Review article › peer-review

2 Citations (Scopus)

Abstract

Hyponatraemia is common in heart failure (HF). It is estimated that over 20% of patients admitted to hospital with HF have hyponatraemia. It has also been repeatedly shown to be a surrogate marker of increased morbidity and mortality in this specific population. This review focuses on the pathophysiology of hyponatraemia through the activation of neurohormonal cascades in HF, the clinical implications of sustained hyponatraemia and treatment options in the management of this challenging phenomenon.

Original language	English
Pages (from-to)	659-666
Number of pages	8
Journal	Internal Medicine Journal
Volume	50
Issue number	6
DOIs	https://doi.org/10.1111/imj.14624
Publication status	Published - Jun 2020

Keywords

acute heart failure
chronic heart failure
hyponatraemia

Access to Document

10.1111/imj.14624Licence: In Copyright

Cite this

@article{90310c5f51da4ef0ac1c74cc74f1c895,

title = "Hyponatraemia in heart failure",

abstract = "Hyponatraemia is common in heart failure (HF). It is estimated that over 20% of patients admitted to hospital with HF have hyponatraemia. It has also been repeatedly shown to be a surrogate marker of increased morbidity and mortality in this specific population. This review focuses on the pathophysiology of hyponatraemia through the activation of neurohormonal cascades in HF, the clinical implications of sustained hyponatraemia and treatment options in the management of this challenging phenomenon.",

keywords = "acute heart failure, chronic heart failure, hyponatraemia",

author = "Tee, {Su Ling} and Andrew Sindone and Simon Roger and John Atherton and John Amerena and Michael D'Emden and Jonathan Erlich and {De Pasquale}, Carmine",

year = "2020",

month = jun,

doi = "10.1111/imj.14624",

language = "English",

volume = "50",

pages = "659--666",

journal = "Internal Medicine Journal",

issn = "0004-8291",

publisher = "Wiley",

number = "6",

}

TY - JOUR

T1 - Hyponatraemia in heart failure

AU - Tee, Su Ling

AU - Sindone, Andrew

AU - Roger, Simon

AU - Atherton, John

AU - Amerena, John

AU - D'Emden, Michael

AU - Erlich, Jonathan

AU - De Pasquale, Carmine

PY - 2020/6

Y1 - 2020/6

N2 - Hyponatraemia is common in heart failure (HF). It is estimated that over 20% of patients admitted to hospital with HF have hyponatraemia. It has also been repeatedly shown to be a surrogate marker of increased morbidity and mortality in this specific population. This review focuses on the pathophysiology of hyponatraemia through the activation of neurohormonal cascades in HF, the clinical implications of sustained hyponatraemia and treatment options in the management of this challenging phenomenon.

AB - Hyponatraemia is common in heart failure (HF). It is estimated that over 20% of patients admitted to hospital with HF have hyponatraemia. It has also been repeatedly shown to be a surrogate marker of increased morbidity and mortality in this specific population. This review focuses on the pathophysiology of hyponatraemia through the activation of neurohormonal cascades in HF, the clinical implications of sustained hyponatraemia and treatment options in the management of this challenging phenomenon.

KW - acute heart failure

KW - chronic heart failure

KW - hyponatraemia

UR - http://www.scopus.com/inward/record.url?scp=85086368281&partnerID=8YFLogxK

U2 - 10.1111/imj.14624

DO - 10.1111/imj.14624

M3 - Review article

C2 - 31449715

AN - SCOPUS:85086368281

SN - 0004-8291

VL - 50

SP - 659

EP - 666

JO - Internal Medicine Journal

JF - Internal Medicine Journal

IS - 6

ER -

Hyponatraemia in heart failure

Abstract

Keywords

Access to Document

Other files and links

Fingerprint

Cite this