Melanopsin knock-out mice have abnormal refractive development and increased susceptibility to form-deprivation myopia

Ranjay Chakraborty; Duk Cheon Lee; Erica G. Landis; Michael A. Bergen; Hanna Park; Curran Sidhu; Samar Hattar; P. Michael Iuvone; Richard A. Stone; Machelle T. Pardue

Melanopsin knock-out mice have abnormal refractive development and increased susceptibility to form-deprivation myopia

Ranjay Chakraborty, Duk Cheon Lee, Erica G. Landis, Michael A. Bergen, Hanna Park, Curran Sidhu, Samar Hattar, P. Michael Iuvone, Richard A. Stone, Machelle T. Pardue

Research output: Contribution to conference › Abstract › peer-review

Abstract

Melanopsin, a photopigment in intrinsically photosensitive retinal ganglion cells (ipRGCs), is involved in regulating visual signaling, retinal dopaminergic cell activity, and circadian activity. Here, we examined the contribution of melanopsin to normal refractive development and to visual form-deprivation (FD) in a mutant mouse lacking melanopsin (Opn4-/-).

Original language	English
Number of pages	2
Publication status	Published - May 2015
Externally published	Yes
Event	The Association for Research in Vision and Ophthalmology annual meeting 2012 - Fort Lauderdale Convention Center, Fort Lauderdale, United States Duration: 6 May 2012 → 10 May 2012 Conference number: 14

Conference

Conference	The Association for Research in Vision and Ophthalmology annual meeting 2012
Abbreviated title	ARVO 2012
Country/Territory	United States
City	Fort Lauderdale
Period	6/05/12 → 10/05/12

Keywords

Melanopsin
abnormal refractive development
mice

Access to Document

https://iovs.arvojournals.org/article.aspx?articleid=2335919&resultClick=1Licence: In Copyright

Cite this

Chakraborty, R., Lee, D. C., Landis, E. G., Bergen, M. A., Park, H., Sidhu, C., Hattar, S., Iuvone, P. M., Stone, R. A., & Pardue, M. T. (2015). Melanopsin knock-out mice have abnormal refractive development and increased susceptibility to form-deprivation myopia. Abstract from The Association for Research in Vision and Ophthalmology annual meeting 2012, Fort Lauderdale, Florida, United States. https://iovs.arvojournals.org/article.aspx?articleid=2335919&resultClick=1

@conference{55284a4d82c24d118460b53221310283,

title = "Melanopsin knock-out mice have abnormal refractive development and increased susceptibility to form-deprivation myopia",

abstract = " Melanopsin, a photopigment in intrinsically photosensitive retinal ganglion cells (ipRGCs), is involved in regulating visual signaling, retinal dopaminergic cell activity, and circadian activity. Here, we examined the contribution of melanopsin to normal refractive development and to visual form-deprivation (FD) in a mutant mouse lacking melanopsin (Opn4-/-).",

keywords = "Melanopsin, abnormal refractive development, mice",

author = "Ranjay Chakraborty and Lee, {Duk Cheon} and Landis, {Erica G.} and Bergen, {Michael A.} and Hanna Park and Curran Sidhu and Samar Hattar and Iuvone, {P. Michael} and Stone, {Richard A.} and Pardue, {Machelle T.}",

year = "2015",

month = may,

language = "English",

note = "The Association for Research in Vision and Ophthalmology annual meeting 2012, ARVO 2012 ; Conference date: 06-05-2012 Through 10-05-2012",

}

Chakraborty, R, Lee, DC, Landis, EG, Bergen, MA, Park, H, Sidhu, C, Hattar, S, Iuvone, PM, Stone, RA & Pardue, MT 2015, 'Melanopsin knock-out mice have abnormal refractive development and increased susceptibility to form-deprivation myopia', The Association for Research in Vision and Ophthalmology annual meeting 2012, Fort Lauderdale, United States, 6/05/12 - 10/05/12. <https://iovs.arvojournals.org/article.aspx?articleid=2335919&resultClick=1>

Melanopsin knock-out mice have abnormal refractive development and increased susceptibility to form-deprivation myopia. / Chakraborty, Ranjay; Lee, Duk Cheon; Landis, Erica G. et al.
2015. Abstract from The Association for Research in Vision and Ophthalmology annual meeting 2012, Fort Lauderdale, Florida, United States.

Research output: Contribution to conference › Abstract › peer-review

TY - CONF

T1 - Melanopsin knock-out mice have abnormal refractive development and increased susceptibility to form-deprivation myopia

AU - Chakraborty, Ranjay

AU - Lee, Duk Cheon

AU - Landis, Erica G.

AU - Bergen, Michael A.

AU - Park, Hanna

AU - Sidhu, Curran

AU - Hattar, Samar

AU - Iuvone, P. Michael

AU - Stone, Richard A.

AU - Pardue, Machelle T.

N1 - Conference code: 14

PY - 2015/5

Y1 - 2015/5

N2 - Melanopsin, a photopigment in intrinsically photosensitive retinal ganglion cells (ipRGCs), is involved in regulating visual signaling, retinal dopaminergic cell activity, and circadian activity. Here, we examined the contribution of melanopsin to normal refractive development and to visual form-deprivation (FD) in a mutant mouse lacking melanopsin (Opn4-/-).

AB - Melanopsin, a photopigment in intrinsically photosensitive retinal ganglion cells (ipRGCs), is involved in regulating visual signaling, retinal dopaminergic cell activity, and circadian activity. Here, we examined the contribution of melanopsin to normal refractive development and to visual form-deprivation (FD) in a mutant mouse lacking melanopsin (Opn4-/-).

KW - Melanopsin

KW - abnormal refractive development

KW - mice

M3 - Abstract

T2 - The Association for Research in Vision and Ophthalmology annual meeting 2012

Y2 - 6 May 2012 through 10 May 2012

ER -

Melanopsin knock-out mice have abnormal refractive development and increased susceptibility to form-deprivation myopia

Abstract

Conference

Keywords

Access to Document

Fingerprint

Cite this