Abstract
Renal tubular acidosis (RTA) is a syndrome resulting from either a defect in proximal tubule bicarbonate (HCO3) reabsorption or a defect in distal tubule hydrogen ion (H·) secretion, or both. This results in normal anion gap hyperchloremic metabolic acidosis (HCMA).
| Original language | English |
|---|---|
| Title of host publication | Pediatric Nephrology |
| Subtitle of host publication | On-the-go |
| Editors | Hui-Kim Yap, Sharon Teo, Kar-Hui Ng |
| Place of Publication | Singapore |
| Publisher | World Scientific Publishing Co., Inc. |
| Pages | 217-235 |
| Number of pages | 19 |
| Edition | 4th |
| ISBN (Electronic) | 978-981-124-825-2, 978-981-124-826-9 |
| ISBN (Print) | 978-981-124-644-9, 978-981-124-851-1 |
| Publication status | Published - 2021 |
| Externally published | Yes |
Keywords
- renal tubular acidosis (RTA)
- proximal tubule bicarbonate
- HCO3
- distal tubule hydrogen ion
- Anion effect
- hyperchloremic metabolic acidosis (HCMA)